Another type of renal failure is called polycystic kidney. Many places now point out that Polycystic Kidney Diseases patients cannot eat rice. What foods can and can't polycystic kidney patients eat? What fruit can polycystic kidney patients eat? So what foods can and can't be eaten by people with PKD?
1. The diet of polycystic kidney patients should be light and avoid spicy and irritating food.
The gastrointestinal mucosa of patients with chronic renal failure, advanced renal failure and uremia is often hyperemic and erosive, such as eating pepper, liquor and other irritating foods, which often aggravates the erosion of gastrointestinal mucosa, leading to blood leakage of gastric endometrial vessels.
2. Dietary patients with polycystic kidney disease should eat soft food, avoid hard food and fried food.
The gastrointestinal mucosa of patients with chronic renal failure is often hyperemia and erosion due to toxin retention in the body and blood microcirculation disturbance. If they eat hard food, fried food such as sesame cake sugar, fried noodles, etc., they often lead to bleeding caused by food cutting blood vessels in the stomach. Because of coagulation dysfunction in patients with chronic renal failure, advanced renal failure and toxicosis, it is very easy to cause death after hemorrhage.
3. Eat more high-calorie foods.
Many data show that the best daily intake of 8368 ~ 12552 kilojoules of calories for patients with renal failure, these high-calorie foods mainly come from fat and sugar. Studies have shown that unsaturated fatty acids in fats and sugars can prevent the deterioration of renal function.
4. Vitamin supplementation.
Chronic renal failure patients often lack water-soluble vitamins and some trace elements such as zinc and iron.
5. Phosphorus limited calcium supplement.
Phosphorus excretion decreases in the early stage of renal failure. Strict restriction of phosphorus intake is not only an effective means to delay the progress of slow renal failure, but also an effective way to reduce the residual renal oxygen consumption, reduce transmembrane pressure difference, alleviate tubulointerstitial damage, reduce the deposition of calcium phosphate in tubules, basement membrane and interstitium, alleviate inflammation damage and reduce the hypermetabolism of renal tubules, which is conducive to delaying renal failure.
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